GLOW (BPC-157 / TB-500 / GHK-Cu Blend)

GHK-Cu + BPC-157 + TB-500 Peptide Blend Research

70mg (10mg / 10mg / 50mg))

Laboratory studies on this triple-peptide blend reveal converging but distinct reparative pathways. GHK-Cu enhances extracellular-matrix remodeling and anti-inflammatory gene expression [1 | 2]. BPC-157 provides broad cytoprotection and gut-centric healing [3], while TB-500 accelerates cell migration and angiogenesis [4 | 5]. Exploring them together lets investigators test whether simultaneous activation of these mechanisms produces additive or synergistic benefits in soft-tissue regeneration, vascular growth, and inflammation control.

Collagen Synthesis & Skin / Wound Healing

• GHK-Cu boosts type I collagen production in cultured fibroblasts at picomolar concentrations [1] and speeds closure of ischemic or diabetic rodent wounds while lowering TNF-α and other pro-inflammatory cytokines [2].
  
• TB-500 further stimulates keratinocyte and endothelial migration into the wound bed through integrin-linked-kinase activation, complementing GHK-Cu’s collagen-driven matrix repair [4 | 5].
  

Gastrointestinal Protection & Systemic Anti-Inflammatory Activity

• BPC-157 reliably prevents and reverses NSAID-, alcohol-, and stress-induced gastric lesions in rats, preserving mucosal integrity and microvascular blood flow [3].
  
• Its normalization of nitric-oxide signaling and dampening of inflammatory mediators may amplify the antioxidant gene programs up-regulated by GHK-Cu [2].
  

Angiogenesis & Tissue Regeneration

• TB-500 is strongly pro-angiogenic, stimulating endothelial chemotaxis, tube formation, and aortic-ring sprouting in vitro and in vivo [5].
  
• Full-length Thymosin-β4 improves cardiomyocyte survival and neovascularization after myocardial infarction, underscoring its broader regenerative potential [4].
  
• GHK-Cu also raises VEGF expression and capillary density in healing tissue, so tandem use could further increase neovascularization in ischemic sites [2].
  

Muscle & Tendon Repair

• BPC-157 accelerates healing of transected Achilles tendons and strengthens regenerated tissue in rats [7]; it also stimulates tenocyte proliferation [8].
  
• TB-500 facilitates actin-cytoskeleton reorganization, essential for myoblast migration and tendon fibroblast alignment during repair [4].
  
• Together with GHK-Cu’s collagen-stimulating actions [1 | 2], the triad enables experiments that measure whether combined treatment shortens functional recovery time or improves tensile strength beyond individual peptide effects.